Estrogen Increases Endothelial Nitric Oxide Synthase via Estrogen Receptors in Rat Cerebral Blood Vessels

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Medroxyprogesterone Acetate or Progesterone Cerebral Blood Vessels : Effect Preserved After Concurrent Treatment With Estrogen Increases Endothelial Nitric Oxide Synthase via Estrogen Receptors in Rat

Medroxyprogesterone Acetate or Progesterone Cerebral Blood Vessels : Effect Preserved After Concurrent Treatment With Estrogen Increases Endothelial Nitric Oxide Synthase via Estrogen Receptors in Rat Print ISSN: 0039-2499. Online ISSN: 1524-4628 Copyright © 2002 American Heart Association, Inc. All rights reserved. is published by the American Heart Association, 7272 Greenville Avenue, Dallas,...

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Estrogen increases endothelial nitric oxide synthase via estrogen receptors in rat cerebral blood vessels: effect preserved after concurrent treatment with medroxyprogesterone acetate or progesterone.

BACKGROUND AND PURPOSE In vivo and in vitro rat models of hormone therapy were used to test the following hypotheses: (1) estrogen acts directly on cerebrovascular estrogen receptors to increase endothelial nitric oxide synthase (eNOS); (2) increased protein correlates with higher NOS activity; and (3) effects of estrogen on eNOS are altered by concurrent treatment with either medroxyprogestero...

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Protein in Rat Cerebral Microvessels Chronic Estrogen Treatment Increases Levels of Endothelial Nitric Oxide Synthase

Protein in Rat Cerebral Microvessels Chronic Estrogen Treatment Increases Levels of Endothelial Nitric Oxide Synthase Print ISSN: 0039-2499. Online ISSN: 1524-4628 Copyright © 1999 American Heart Association, Inc. All rights reserved. is published by the American Heart Association, 7272 Greenville Avenue, Dallas, TX 75231 Stroke doi: 10.1161/01.STR.30.10.2186 1999;30:2186-2190 Stroke. http://st...

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Chronic estrogen treatment increases levels of endothelial nitric oxide synthase protein in rat cerebral microvessels.

BACKGROUND AND PURPOSE A number of studies indicate that the female gonadal hormone, estrogen, confers protection against cerebrovascular disorders such as stroke. One postulated mechanism for these effects of estrogen is an action on the enzyme endothelial nitric oxide synthase (eNOS), which produces the vasodilatory molecule NO. We have investigated the hypothesis that estrogen increases expr...

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Endothelial nitric oxide synthase and estrogen.

See article by M.R. Andersen and S. Stender [1] (pages arginine to L-citrulline and nitric oxide (NO). Since NO 192 –199) in this issue. and citrulline are formed in equi-molar quantities, NO formation was expressed in terms of citrulline production Andersen and Stender [1] have reported regional variaper million endothelial cells. Thus it was possible to tions in endothelial nitric oxide synth...

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ژورنال

عنوان ژورنال: Stroke

سال: 2002

ISSN: 0039-2499,1524-4628

DOI: 10.1161/01.str.0000016325.54374.93